249. Ismail, W. I. W.; Abu, M. N.; Salleh, M. A. M.; Radzman, N. H. M.; Yusof, R. M.; Hassan, H. F. Insulin sensitivity enhancement of the mixture of Tinospora crispa and gelam (Melaleucacajuputi) honey and its antiproliferative activity on hepatocellular carcinoma, HepG2: A preliminary study. J Med Res Dev 2013; 2: 48–54.

250. Chanchao, C. Bioactivity of honey and propolis of Tetragonula Laeviceps in Thailand. In Bioactivity of honey and propolis of Tetragonula Laeviceps in Thailand.; In Pot-Honey. Springer: New York, NY, USA, 2013; pp. 495–505.

251. El-kott, A. F.; Kandeel, A. A.; El-Aziz, S. F. A.; Ribea, H.M. Anti-tumor effects of bee honey on PCNA and P53 expression in the rat hepatocarcinogenesis. Int J Cancer Res 2012; 8: 130–139.

252. Wen, C. T.; Hussein, S. Z.; Abdullah, S.; Karim, N. A.; Makpol, S.; Mohd, Y. A. Gelam and Nenas honeys inhibit proliferation of HT 29 colon cancer cells by inducing DNA damage and apoptosis while suppressing inflammation. Asian Pac J Cancer Prev 2012; 13: 1605–1610.

253. Jaganathan, S.; Mandal, M. Honey constituents and its apoptotic effect in colon cancer cells. Apiprod Apimed Sci 2009; 1: 29–36.

254. Oršolić, N.; Terzić, S.; Šver, L.; Bašić, I. Honey-bee products in prevention and/or therapy of murine transplantable tumours. J. Sci. Food Agric. 2005; 85: 363–370.

255. Orsolic, N.; Basic, I. Honey as a cancer-preventive agent. Period. Biol. 2004; 106: 397–401.

256. Mabrouk, G. M.; Moselhy, S. S.; Zohny, S. F.; Ali, E. M.; Helal, T. E.; Amin, A. A.; Khalifa, A. A. Inhibition of methylnitrosourea (MNU) induced oxidative stress and carcinogenesis by orally administered bee honey and Nigella grains in Sprague Dawely rats. J Exp Clin Cancer Res 2002; 21: 341–346.

257. Tsiapara, A. V.; Jaakkola, M.; Chinou, I.; Graikou, K.; Tolonen, T.; Virtanen, V.; Moutsatsou, P. Bioactivity of Greek honey extracts on breast cancer (MCF-7), prostate cancer (PC-3) and endometrial cancer (Ishikawa) cells: Profile analysis of extracts. Food Chem. 2009; 116: 702–708.

258. Samarghandian, S.; Afshari, J. T.; Davoodi, S. Honey induces apoptosis in renal cell carcinoma. Pharmacogn. Mag. 2011; 7: 46–52.

259. Pichichero, E.; Cicconi, R.; Mattei, M.; Muzi, M. G.; Canini, A. Acacia honey and chrysin reduce proliferation of melanoma cells through alterations in cell cycle progression. Int. J. Oncol. 2010; 37: 973–981.

260. Aliyu, M.; Odunola, O. A.; Farooq, A. D.; Rasheed, H.; Mesaik, A.M.; Choudhary, M.I.; Channa, I.S.; Khan, S. A.; Erukainure, O. L. Molecular mechanism of antiproliferation potential of Acacia honey on NCI-H460 cell line. Nutr. Cancer 2013; 65: 296–304.

261. Ghashm, A. A.; Othman, N. H.; Khattak, M. N.; Ismail, N. M.; Saini, R. Antiproliferative effect of tualang honey on oral squamous cell carcinoma and osteosarcoma cell lines. BMC Complement Altern Med 2010; 10: 49.

262. Morales, P.; Haza, A. I. Antiproliferative and apoptotic effects of spanish honeys. Pharmacogn Mag 2013; 9: 231–237.

263. Ghaffari, A.; Somi, M. H.; Safaiyan, A.; Modaresi, J.; Ostadrahimi, A. Honey and apoptosis in human gastric mucosa. Health Promot. Perspect. 2012; 2: 53–59.

264. Erejuwa OO1, Sulaiman SA2, Wahab MS3. Effects of honey and its mechanisms of action on the development and progression of cancer. Molecules. 2014 Feb 21;19(2):2497–522.

265. National Institute of Health State-of-the-science conference statement: multivitamin/mineral supplements and chronic diseae prevention. Ann Intern Med 2006;145:364.

266. Lin J, Cook NR, Albert C, et al. Vitamins C and E and beta-carotene supplementation and cancer risk: a randomized controlled trial. J Natl Cancer Inst 2009; 101: 14.

267. Qiao YL, Dawsey SM, Kamangar F, Fan JH, Abnet CC, Sun XD, Johnson LL, Gail MH, Dong ZW, Yu B, Mark SD, Taylor PR. Total and cancer mortality after supplementation with vitamins and minerals: follow-up of the Linxian General Population Nutrition Intervention Trial. J Natl Cancer Inst. 2009 Apr 1; 101(7): 507–18.

268. Larsson SC, Hakansson N, Giovanucci E, Wolk A. Folate intake and pancreatic cancer incidence: a prospective study of Swedish women and men. J Natl Cancer Inst 2006; 15; 98(6): 407–13.

269. Larsson SC, Giovanucci E, Wolk A. Folate intake, MTHFR polymorphisms, and risk of esophageal, gastric, and pancreatic cancer: a meta-analysis. Gastroenterology 2006;131:1271.

270. Knekt P, Reunanen A, Takkunen H, et al. Body iron stores and risk of cancer. Int J Cancer 1994; 56:379.

271. Zacharski LR, Chow BK, Howes PS, et al. Decreased cancer risk after iron reduction in patients with peripheral arterial disease: results from a randomized trial. J Natl Cancer Inst 2008; 100: 996.

272. Genkinger JM, Wang M, Li R, Albanes D, Anderson KE, Bernstein L, van den Brandt PA, English DR, Freudenheim JL, Fuchs CS, Gapstur SM, Giles GG, Goldbohm RA, Håkansson N, Horn-Ross PL, et al. Dairy products and pancreatic cancer risk: a pooled analysis of 14 cohort studies. Ann Oncol. 2014 Jun; 25(6): 1106–15.

273. Nazir L, Samad F, Haroon W, Kidwai SS, Siddiqi S, Zehravi M. Comparison of glycaemic response to honey and glucose in type 2 diabetes. J Pak Med Assoc. 2014 Jan; 64(1): 69–71.

274. Nasrolahi O, Heidari R, Rahmani F, Farokhi F. Effect of natural honey from Ilam and metformin for improving glycemic control in streptozotocin-induced diabetic rats. Avicenna J Phytomed 2012 Fall; 2(4): 212–21.

275. Abdulrhman MM, El-Hefnawy MH, Aly RH, Shatla RH, Mamdouh RM, Mahmoud DM, Mohamed WS. Metabolic effects of honey in type 1 diabetes mellitus: a randomized crossover pilot study. J Med Food. 2013 Jan; 16(1): 66–72.

276. Sanz ML, Polemis N, Morales V, Corzo N, Drakoularakou A, Gibson GR, Rastall RA. In vitro investigation into the potential prebiotic activity of honey oligosaccharides. J Agri Food Chem. 2005; 53: 2914–2921.

277. Markelov VV, Trushin MV. Bee venom therapy and low dose naltrexone for treatment of multiple sclerosis. Nepal J Neurosci 2006; 3: 71–77.

278. Simopoulos AP. The importance of the ratio of omega-6/omega-3 essential fatty acids. Biomed Pharmacother. 2002 Oct; 56(8): 365–79.

Чай является вторым наиболее часто употребляемым напитком после воды(1), и особенно популярен в азиатских странах. Чай, полученный из растения Camelia sinensis, классифицируется как черный, зеленый и оолонг. Зеленый чай классифицируется на «белый», «желтый» и собственно «зеленый». Последний меньше всего подвергается процессу ферментации при обработке.